Update bibliography

_bibliography/papers.bib

@@ -20,17 +20,21 @@ @article{dzyubenko_role_2018
 	pdf = {Dzyubenko et al. - 2018 - Role of immune responses for extracellular matrix .pdf},
 }
 
-@article{einstein1950meaning,
-  abbr={AJP},
-  bibtex_show={true},
-  title={The meaning of relativity},
-  author={Einstein, Albert and Taub, AH},
-  journal={American Journal of Physics},
-  volume={18},
-  number={6},
-  pages={403--404},
-  year={1950},
-  publisher={American Association of Physics Teachers}
+@misc{manrique-castano_dissecting_2023,
+	title = {Dissecting glial scar formation by spatial point pattern and topological data analysis},
+	copyright = {© 2023, Posted by Cold Spring Harbor Laboratory. This pre-print is available under a Creative Commons License (Attribution 4.0 International), CC BY 4.0, as described at http://creativecommons.org/licenses/by/4.0/},
+	url = {https://www.biorxiv.org/content/10.1101/2023.10.04.560910v1},
+	doi = {10.1101/2023.10.04.560910},
+	abstract = {Glial scar formation represents a fundamental response to central nervous system (CNS) injury. It is mainly characterized by a well-defined spatial rearrangement of reactive astrocytes and microglia. The mechanisms underlying glial scar formation have been extensively studied, yet quantitative descriptors of the spatial arrangement of reactive glial cells remain limited. Here, we present a novel approach using point pattern analysis (PPA) and topological data analysis (TDA) to quantify spatial patterns of reactive glial cells after experimental ischemic stroke in mice. We provide open and reproducible tools using R and Julia to quantify spatial intensity, cell covariance and conditional distribution, cell-to-cell interactions, and short/long-scale arrangement, which collectively disentangle the arrangement patterns of the glial scar. This approach unravels a substantial divergence in the distribution of reactive astrocytes and microglia after injury that conventional analysis methods cannot fully characterize. PPA and TDA are valuable tools for studying the complex spatial arrangement of reactive glia and other nervous cells following CNS injuries and have potential applications for evaluating glial-targeted restorative therapies.},
+	language = {en},
+	urldate = {2024-03-01},
+	publisher = {bioRxiv},
+	author = {Manrique-Castano, Daniel and Bhaskar, Dhananjay and ElAli, Ayman},
+	month = oct,
+	year = {2023},
+	note = {Pages: 2023.10.04.560910
+Section: New Results},
+	pdf = {Manrique-Castano et al. - 2023 - Dissecting glial scar formation by spatial point.pdf},
 }
 
 @article{dzyubenko_topological_2018,
@@ -48,21 +52,27 @@ @article{dzyubenko_topological_2018
   doi={10.1016/j.matbio.2018.08.001},
   url={https://linkinghub.elsevier.com/retrieve/pii/S0945053X18301872},
   html={https://linkinghub.elsevier.com/retrieve/pii/S0945053X18301872},
-  pdf={example_pdf.pdf},
+  pdf={Dzyubenko et al. - 2018 - Topological remodeling of cortical perineuronal ne},
   dimensions={true},
   google_scholar_id={Tyk-4Ss8FVUC},
   selected={true}
 }
 
-@article{einstein1905molekularkinetischen,
-  title={{\"U}ber die von der molekularkinetischen Theorie der W{\"a}rme geforderte Bewegung von in ruhenden Fl{\"u}ssigkeiten suspendierten Teilchen},
-  author={Einstein, A.},
-  journal={Annalen der physik},
-  volume={322},
-  number={8},
-  pages={549--560},
-  year={1905},
-  publisher={Wiley Online Library}
+@article{dzyubenko_tenascin-c_2022,
+	title = {Tenascin-{C} restricts reactive astrogliosis in the ischemic brain},
+	volume = {110},
+	issn = {0945-053X},
+	url = {https://www.sciencedirect.com/science/article/pii/S0945053X22000555},
+	doi = {10.1016/j.matbio.2022.04.003},
+	abstract = {Cellular responses in glia play a key role in regulating brain remodeling post-stroke. However, excessive glial reactivity impedes post-ischemic neuroplasticity and hampers neurological recovery. While damage-associated molecular patterns and activated microglia were shown to induce astrogliosis, the molecules that restrain astrogliosis are largely unknown. We explored the role of tenascin-C (TnC), an extracellular matrix component involved in wound healing and remodeling of injured tissues, in mice exposed to ischemic stroke induced by transient intraluminal middle cerebral artery occlusion. In the healthy adult brain, TnC expression is restricted to neurogenic stem cell niches. We previously reported that TnC is upregulated in ischemic brain lesions. We herein show that the de novo expression of TnC post-stroke is closely associated with reactive astrocytes, and that astrocyte reactivity at 14 days post-ischemia is increased in TnC-deficient mice (TnC−/−). By analyzing the three-dimensional morphology of astrocytes in previously ischemic brain tissue, we revealed that TnC−/− reduces astrocytic territorial volume, branching point number, and branch length, which are presumably hallmarks of the homeostatic regulatory astrocyte state, in the post-acute stroke phase after 42 days. Interestingly, TnC−/− moderately increased aggrecan, a neuroplasticity-inhibiting proteoglycan, in the ischemic brain tissue at 42 days post-ischemia. In vitro in astrocyte-microglia cocultures, we showed that TnC−/− reduces the microglial migration speed on astrocytes and elevates intercellular adhesion molecule 1 (ICAM1) expression. Post-stroke, TnC−/− did not alter the ischemic lesion size or neurological recovery, however microglia-associated ICAM1 was upregulated in TnC−/− mice during the first week post stroke. Our data suggest that TnC plays a central role in restraining post-ischemic astrogliosis and regulating astrocyte-microglial interactions.},
+	urldate = {2024-03-01},
+	journal = {Matrix Biology},
+	author = {Dzyubenko, Egor and Manrique-Castano, Daniel and Pillath-Eilers, Matthias and Vasileiadou, Paraskevi and Reinhard, Jacqueline and Faissner, Andreas and Hermann, Dirk M},
+	month = jun,
+	year = {2022},
+	keywords = {Astrocyte morphology, Extracellular matrix, Focal cerebral ischemia, Glial scar, Microglia, Tenascin C},
+	pages = {1--15},
+	pdf = {Tenascin-C restricts reactive astrogliosis in the ischemic brain.pdf}
 }
 
 @article{einstein1905movement,